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development of IAA was thought to be probably as a consequence of oxidase deamination of NMT (Barker et al., 1980), but was afterwards established to become also partly by direct deamination by MAO (Barker et al., 1982). Pretreatment Together with the MAO-I iproniazid in rat full Mind homogenates inhibited IAA development by 83%, NMT and DMT-NO for
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